When Prolotherapy Fails
Although prolotherapy is extremely effective at treating pain and injuries, it can on occasion fail to deliver optimal results. The success of prolotherapy treatments depends on several factors with the most important being the skill of the prolotherapist. The treatment is completely dependent on how skilled the doctor is at assessing and injecting the correct structures causing the pain and dysfunction. Thus, an inexperienced prolotherapist may hinder the success of your prolotherapy treatment.
Also, if the area isn’t treated completely (with enough injections), with a strong enough solution or with an adequate volume of solution, then the patient will not receive the full benefit of prolotherapy because of insufficient inflammation. Remember, the body heals by inflammation. A person should feel stiffness, tightness and soreness for at least one to two days following a prolotherapy treatment. If they don’t, then the immune reaction may not be strong enough to regenerate enough connective tissue needed to heal the structures treated. In this case, the physician needs to investigate the reason for the poor immune response.
Usually most people are cured of their pain with three to six Prolotherapy sessions. If a patient continues to suffer from pain or there is not significant improvement, and assuming that the correct structures are being treated with adequate inflammation, chances are that there is an underlying cause that hasn’t been identified.
Below is a list of factors that may prevent affective healing of connective tissue thus causing dissapointing results after prolotherapy treatments. It becomes important that any deficiencies and illnesses be corrected in order to strengthen the immune system to allow the body to respond to Prolotherapy.
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Factors
Affecting Healing of |
|
|
Age |
Overall health status ie: chronic illness |
|
Lack of blood Supply/poor circulation ie: smokers, Atherosclerosis, hypothyroid |
pH and lactate concentration ie: too acidic |
|
Degree of hypoxia (systemic and local) |
Pharmacological agents (drugs) ie: steroids |
|
Dietary intake ie: poor diet |
Psychological
influences (placebo |
|
Electrical fields |
Severity of injury |
|
Growth factors, cytokines, eicosanoids ie: EFA deficiency |
Structural (physical) deformities |
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Hormonal influences ie: elevated estrogen, decreased growth hormone, DHEA testosterone and progesterone |
Temperature |
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Mechanical load forces |
Timing and return to physical activity |
|
Metabolic
and cell turnover rates of |
Type of injury |
|
Mobility (local & whole body) |
Type of onset (acute or chronic) |
|
Muscular strength and forces |
Type of tissue(s) affected |
|
Nutritional status ie: poor digestion, food allergies |
Underlying disease processes |